Mitochondria as Targets for Endothelial Protection in COVID-19

B. Chernyak, E. Popova, L. Zinovkina, K. Lyamzaev, R. Zinovkin,

2021

https://doi.org/10.3389/fphys.2020.606170

Цитирование

Об авторах

B. Chernyak
A.N. Belozersky Institute of Physico-Chemical Biology, M.V. Lomonosov Moscow State University, Moscow, Russian Federation
E. Popova
Faculty of Bioengineering and Bioinformatics, M.V. Lomonosov Moscow State University, Moscow, Russian Federation
L. Zinovkina
Institute of Molecular Medicine, Sechenov First Moscow State Medical University, Moscow, Russian Federation
K. Lyamzaev
R. Zinovkin

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Ключевые слова

angiotensin II; cryopyrin; endothelial leukocyte adhesion molecule 1; immunoglobulin enhancer binding protein; inducible nitric oxide synthase; intercellular adhesion molecule 1; interferon; interleukin 1beta; interleukin 6; mitogen activated protein kinase; reactive oxygen metabolite; reduced nicotinamide adenine dinucleotide phosphate oxidase; reduced nicotinamide adenine dinucleotide phosphate oxidase 2; vascular cell adhesion molecule 1; viral protein; Article; cell activation; cell damage; cell membrane permeability; coronavirus disease 2019; endothelium cell; human; infection prevention; inflammation; mitochondrion; nonhuman; protein degradation; protein expression; protein interaction; protein localization; Severe acute respiratory syndrome coronavirus 2; signal transduction

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Тип лицензии Creative Commons

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